Evidence for a connection between coronavirus disease-19 and exposure to radiofrequency radiation from wireless communications including 5G
Quote:Table 1 lists the manifestations common to COVID-19 including disease progression and the corresponding adverse bioeffects from WCR exposure. Although these effects are delineated into categories — blood changes, oxidative stress, immune system disruption and activation, increased intracellular calcium (Ca2+), and cardiac effects — it must be emphasized that these effects are not independent of each other. For example, blood clotting and inflammation have overlapping mechanisms, and oxidative stress is implicated in erythrocyte morphological changes as well as in hypercoagulation, inflammation, and organ damage.
Table 1
Bioeffects of Wireless Communication Radiation (WCR) exposure in relation to COVID-19 manifestations and their progression
a) Wireless communications radiation (WCR) exposure bioeffects
b) COVID-19 manifestations
a) Blood changes
Short-term: rouleaux, echinocytes
Long-term: reduced blood clotting time, reduced hemoglobin, hemodynamic disorders
b) Blood changes
Rouleaux, echinocytes
Hemoglobin effects; vascular effects
→Reduced hemoglobin in severe disease; autoimmune hemolytic anemia; hypoxemia and hypoxia
→Endothelial injury; impaired microcirculation; hypercoagulation; disseminated intravascular coagulopathy (DIC); pulmonary embolism; stroke
a) Oxidative stress
Glutathione level decrease; free radicals and lipid peroxide increase; superoxide dismutase activity decrease; oxidative injury in tissues and organs
b) Oxidative stress
Glutathione level decrease; free radical increase and damage; apoptosis→Oxidative injury; organ damage in severe disease
a) Immune system disruption and activation
Immune suppression in some studies; immune hyperactivation in other studies
Long-term: suppression of T-lymphocytes; inflammatory biomarkers increased; autoimmunity; organ injury
b) Immune system disruption and activation
Decreased production of T-lymphocytes; elevated inflammatory biomarkers.
→Immune hyperactivation and inflammation; cytokine storm in severe disease; cytokine-induced hypo-perfusion with resulting hypoxia; organ injury; organ failure
a) Increased intracellular calcium
From activation of voltage-gated calcium channels on cell membranes, with numerous secondary effects
b) Increased intracellular calcium
→Increased virus entry, replication, and release
→Increased NF-κB, pro-inflammatory processes, coagulation, and thrombosis
a) Cardiac effects
Up-regulation of sympathetic nervous system; palpitations and arrhythmias
b) Cardiac effects
Arrhythmias
→Myocarditis; myocardial ischemia; cardiac injury; cardiac failure